LRRK2/LRRK1 interactions modulate Rab7 activity and inhibit lysosomal exocytosis
Authors Merghani M, Gerhardt E, Hesse M, Fahlbusch C, Boecker CA, Outeiro TF Journal BioRxiv Citation bioRxiv 2026.06.12.731951. Abstract Mutations in the LRRK2 gene are the most common genetic cause of both familial and sporadic Parkinson’s disease (PD). LRRK2 belongs to the leucine-rich repeat kinase (LRRK) family. Two members of


