Susann Meyer – Page 17

08.07.2025

Nucleic Acids Res

08.07.2025

FASTKD5 processes mitochondrial pre-mRNAs at noncanonical cleavage sites

Antonicka H, Vučković A, Weraarpachai W, Hong S, Brischigliaro M, Ahn A, Barrientos A, Hillen HS, Shoubridge EA

04.07.2025

Sci Adv

04.07.2025

CryoRhodopsins: A comprehensive characterization of a group of microbial rhodopsins from cold environments

Lamm GHU, Marin E, Alekseev A, Schellbach AV, Stetsenko A, Haro-Moreno JM, Bourenkov G, Borshchevskiy V, Asido M, Agthe M, Engilberge S, Rose SL, Caramello N, Royant A, Schneider TR, Bateman A, Mager T, Moser T, Rodriguez-Valera F, Wachtveitl J, Guskov A, Kovalev K

03.07.2025

Dis Model Mech

03.07.2025

Lamin variants cause cardiac arrhythmogenicity in Drosophila

van Wijk SW, Vree P, Huiskes FG, van der Palen RL, Liutkute A, Voigt N, Wallrath LL, Brundel BJJM

01.07.2025

Chemistry

01.07.2025

Fluorinated Phenylazopyrazoles for Monitoring the Photoisomerization in Complex Systems

Jakob L, van der Wal JJ, Tovtik R, Copko J, Iagatti A, Donato MD, Crespi S, Simeth NA

27.06.2025

Calcium channels of synapses of sensory hair cells couple the sound stimulus to the excitation of the auditory nerve. Compared to the intact CaV1.3 channel (wildtyp, left), the genetically modified CaVAG variant, which is based on a minimal change in the blueprint of the CACNAD1 gene, causes activation of the synapses of sensory hair cells in response to very weak sound stimuli and thus leads to increased auditory nerve activity. The mouse model shows that the increased voltage sensitivity of the channel results in damage to the synapses due to overexcitation even at everyday sound levels. Figure: Karagulyan/Moser.

27.06.2025

Calcium channel in the ear: How a tiny mistake affects hearing

Researchers at the University Medical Center Göttingen (UMG) and the Göttingen Cluster of Excellence “Multiscale Bioimaging” (MBExC) have shown how a minimal change in a single ion channel increases the […]

27.06.2025

Calcium channels of synapses of sensory hair cells couple the sound stimulus to the excitation of the auditory nerve. Compared to the intact CaV1.3 channel (wildtyp, left), the genetically modified CaVAG variant, which is based on a minimal change in the blueprint of the CACNAD1 gene, causes activation of the synapses of sensory hair cells in response to very weak sound stimuli and thus leads to increased auditory nerve activity. The mouse model shows that the increased voltage sensitivity of the channel results in damage to the synapses due to overexcitation even at everyday sound levels. Figure: Karagulyan/Moser.

27.06.2025

Calcium channel in the ear: How a tiny mistake affects hearing

Researchers at the University Medical Center Göttingen (UMG) and the Göttingen Cluster of Excellence “Multiscale Bioimaging” (MBExC) have shown how a minimal change in a single ion channel increases the […]